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VIAGRA AND TESTOCREME

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Viagra and Testocreme Together

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VIAGRA in monterey, CA

In comparing erection enhancers and TestoCreme®

It is important to note they make a perfect team. Testosterone (T) has been used topically to treat low testosterone levels for decades. However, it is important to distinguish that the stimulation of libido by testosterone is essential for medications such as Sildenafil, (Viagra®) OR Tadalafil, (Cialis®) to exert their erection enhancement effect. In many cases, both compounds used simultaneously will provide a synergistic effect. What follows is a quick review of the literature to support the hypothesis that both Viagra® and TestoCreme® used together have a positive action in improving the quality of erections and enjoyment for men during intercourse.

Erectile Dysfunction (ED) affects about 30 million men in the United States. The available treatments for ED have undergone a steady evolution in the past 25 years toward less invasive modalities. These treatments have developed in parallel with the understanding of the pathophysiologic mechanisms at work in ED. The introduction of the oral erection enhancers, of other oral generic agents such as Tadalafil and Sildenafil, both-5 phosphodiesterase inhibitors, perhaps represents the culmination of this metamorphosis. The ease of administration of these agents is appealing to a broad sector of men with ED. Oral Sildenafil® therapy provides results comparable to those of other available ED treatment modalities, for example: penis pumps, confidence rings, and urethral inserts.

Testosterone Effect on Aging Men

The use of Viagra® can augment the action of lower testosterone levels. Aging men develop a significant loss of muscle strength that occurs in conjunction with a decline in serum testosterone concentrations. Increasing testosterone concentrations in older men increases skeletal muscle protein synthesis and strength. This increase may be mediated by stimulation of the intramuscular IGF-I system.There is considerable interest in the relationship between testosterone and sexual behavior in men, but the few available data bearing on this issue are inconclusive. Testosterone concentration did not correlate with the sexual activity and interest variables. These results provide evidence that differences among men in circulating testosterone concentration within the normal range do not account for differences in sexual activity and interest. It is also unlikely that variations in sexual activity account for differences in testosterone concentration. It appears that a factor more sensitive than total testosterone levels can be used as a marker for ED.

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DHT and Sex Drive

Dihydrotestosterone is capable of maintaining sex functions in hypogonadal men. There is no evidence that androgen administration in excess of the individually determined critical levels further enhances sex functions. In view of the rapidly declining blood levels of androgens with the available parenteral testosterone ester preparations, the results suggest that hypogonadal patients may benefit from a more frequent administration of these preparations. However, if it is the conversion of testosterone to DHT which stimulates libido, then any increase in activity of the enzyme which converts testosterone to higher levels of DHT will stimulate sexual desire.
Androgens are essential for the expression of normal libido in males, but their role in the maintenance of the erectile response in humans is controversial. In the rat, castration induces loss of penile reflexes and a considerable reduction in the erectile response to electric field stimulation (EFS) of the cavernosal nerve.
Both effects can be reversed by testosterone replacement. Castration reduced the EFS-induced erectile response by 50% in comparison with intact rats, and testosterone restored this decrease to normal. When finasteride was given to these testosterone-treated castrate rats, the erectile response was not restored. DHT was as effective as testosterone in restoring response to EFS in castrates, and this effect was not decreased by finasteride. Nitric oxide synthase activity in the penile body was measured by the arginine-citrulline conversion and was found to correlate with the EFS determinations. These results show that DHT is the active androgen in the prevention of erectile failure seen in castrated rats and suggest that this effect may be mediated, at least partially, by changes in nitric oxide synthase levels in the penis.

In summary

Transdermal testosterone has been found to be both safe and efficacious for hormone replacement or supplementation for men who are deficient. The use of TestoCreme® transdermal testosterone-based cream offers a simple alternative method of treating hypogonadism TestoCreme® has been shown to be both effective in raising free and total T and is more convenient for patients than other methods of testosterone delivery.

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